The R -gene mediated, hypersensitive-type resistance (HR, incompatible interaction) is based on a specific interaction, either directly or indirectly, of a bacterial effector gene product with the R gene of the host plant
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It has also been shown that disulfide-thiol rearrangements within envelopes can cause conformational changes of the peptide, allowing for entry of a virus into a cell [6]
We are continually exploring new models for assessing binding site abilities, with lysine poised to become the next research hotspot among targeted amino acids due to its numerous protein binding sites and extracellular locations
Oxidative Medicine and Cellular Longevity, 2012 , 324832