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KEAP1 acts as an OS sensor to regulate NRF2 turnover within the cell/body, while NRF2 activation leads to the production of antioxidant and detoxifying substances that decrease OS [66,67,68]

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Remarkably, treatment of aged fibroblasts with GHK-Cu shifted their gene expression profiles toward patterns resembling younger cells, suggesting potential for reversing aspects of cellular aging

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