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Mechanistic analyses revealed that bacterial inactivation was associated to hydroxyl radical generation, and electrostatic interactions between positively charged N-CQDs and negatively charged bacterial membranes, leading to morphological changes and cell wall damage [133]

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Peptides add cellular protection, anti-inflammatory effects, tissue repair mechanisms

Metabolic control of the scaffold protein TKS5 in tissue-invasive, proinflammatory T cells

adding GIP agonism appears to further enhance the appetite and metabolic effect

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