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The response to hypoxic conditions involves several compensatory mechanisms initiated through hypoxia-inducible factors and other signaling pathways 6

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These growth factors promote endothelial cell migration and new blood vessel formation, addressing the critical vascular requirement for bone healing

On the other hand, iron overload induced ferroptosis in foam cells which led to plaque instability, and ferroptosis in foam cells increased IL-1 and IL-8 expression, promoting inflammatory responses (Su et al., 2021)

Phospholipid-polymer hybrid nanoparticle-mediated transfollicular delivery of quercetin: prospective implement for the treatment of androgenic alopecia