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The GSSG/GSH ratio in the ALS group was also significantly higher than that in the HC group at the second visit (2.84 vs 1.33, P = 0.0120)

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Conversely, the observed rise in EGT levels in gliK 18 , deficient in gliotoxin biosynthesis, could be due to either a sensory deficiency in the cellular oxidative stress response, or a compensatory mechanism to replace the frontline antioxidant, GSH, utilised for gliotoxin biosynthesis but which cannot undergo replenishment due to GliK -glutamyl cyclotransferase deficiency

On the other hand, paracetamol did not influence convulsion or death that may be induced by pentetrazol (7), quinolones (8) or febrile seizures (9) or alternatively, it prevented pentylenetetrazol-induced seizures (10)

doi: 10.5740/jaoacint.18-0418