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High susceptibility to infection with a variety of bacterial genera, including Haemophilus , has been described in several iron overload disorders, such as hereditary haemochromatosis, -thalassemia, sideroblastic anaemia, transfusion-dependence, or chronic liver disease (Weinberg, 2000

It crosses the blood-brain barrier and activates the hepatocyte growth factor (HGF) / c-Met receptor pathway, a signaling system that governs neuron survival, synaptic formation, and neural circuit repair

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While autophagy has been implicated in Wilsons disease [2], the roles of inflammation and oxidative stress have been debated [13, 14]