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Lanting C, De Kleine E, Van Dijk P

Furthermore, we confirm that the initial glutathionylation of the glutaredoxin moiety and not the glutathione transfer to the roGFP2 moiety is rate-limiting for the oxidation of fusion constructs at physiological or low micromolar GSSG concentrations, thus allowing glutaredoxin structure-function analysis in vivo

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In this pathway, GPX4 also serves as a crucial regulator of ferroptosis

A novel recognition motif for phosphatidylinositol 3-kinase binding mediates its association with the hepatocyte growth factor/scatter factor receptor

Transcriptional change in a cultured cell line is a hypothesis-generating observation