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The pathophysiology of hypertension involves a complex interaction of multiple vascular effectors including the activation of the sympathetic nervous system, of the reninangiotensinaldosterone system and of the inflammatory mediators

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When gut inflammation disrupts tryptophan metabolism, shunting it toward the kynurenine pathway instead of serotonin synthesis, the brain experiences a serotonin deficit even when dietary tryptophan intake is adequate

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