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The toxic effects of AFB1 arise through its enzymatic activation by cytochromes P450 to form highly reactive AFB1-8,9-epoxides

Tissue-threshold split receptor density, downstream signal amplification, and per-tissue glucagon sensitivity differ across liver, visceral fat, sinus node, and subcutaneous fat

Peptides can alter the potency of other agents

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The application of both drugs contributed to a considerable reduction in tumor volume in mice, decreasing from 100 mm3 to less than 3 mm3 after 21 days [139]