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A 2024 review evaluated the molecular mechanisms of biotin in modulating inflammatory diseases

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Roemer and Krysan, 2014)

ROS and oxidatively modified macromolecules function as damage-associated molecular patterns (DAMPs), engaging pattern recognition receptors (PRRs) on microglia, including Toll-like receptors (TLRs) and NOD-like receptors (NLRs), to initiate inflammatory cascades ( Microglia, the resident immune cells of the CNS, are tightly regulated under physiological conditions to maintain neural homeostasis

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Your body releases it from extracellular matrix proteins whenever tissue damage occurs, essentially functioning as a biological signal that activates repair mechanisms