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Results Tobacco-free and regular cigarette smoke exposure produce similar effects on leucocyte influx but not on the BALF inflammatory cell profile Smoke exposure to both TFC and RC increased the inflammatory cell influx to lung parenchyma, reflected in a higher BALF leucocyte cell count, compared to the control group [F (2, 21) = 32.89

These toxic compounds in vivo can decrease endogenous antioxidants, increase lipid peroxidation and oxidative stress, and elevate pro-inflammatory factor concentrations in the blood of the host (van der Vaart et al., 2004

Table 4 presents a logistic regression analysis examining predictors of smoking cessation, including demographic variables, baseline cigarette consumption, years of smoking, COex, plasma cotinine, and group assignment (active vs Sham dTMS)

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