Baines CP, Kaiser RA, Purcell NH, Blair NS, Osinska H, Hambleton MA, Brunskill EW, Sayen MR, Gottlieb RA, Dorn GW, Robbins J, Molkentin JD
Insulin-stimulated glucose uptake by skeletal muscle cells is accounted for by the increased amount of GLUT4 in plasma membrane, known as the sarcolemma in skeletal muscle cells (predominantly the T-tubule membrane fraction), and is not due to changes in the kinetic characteristics of the transporter
Make it automatic rather than something requiring daily decision-making
Some of the negative effects of high homocysteine levels are modulated by the PON1 genotype.[ref] BLMH and BPHL are additional enzymes that can also metabolize homocysteine-thiolactone.[ref] Higher levels of homocysteine-thiolactone are also found in chronic inflammatory conditions, such as rheumatoid arthritis, inflammatory eye disease, and AMD.[ref][ref][ref] Lets take a look at why high homocysteine levels cause problems in the body, and then well look at genetic variants in the above cycles that can affect homocysteine levels when combined with dietary choices
Stock C
That last category is the one that would settle the debate, and right now it remains empty
