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TLR4 is responsive to bacterially derived lipopolysaccharide (LPS) which is an endotoxin secreted by gram-negative bacteria

Insulin-like growth factor I interfaces with brain-derived neurotrophic factor-mediated synaptic plasticity to modulate aspects of exercise-induced cognitive function

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There are hundreds of different mediators, including histamine, heparin (involved in clot prevention), proteases (enzymes that break down proteins), prostaglandins, leukotrienes, a range of cytokines and chemokines (immune signaling molecules), and more (3)

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