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In fact, the CntA / B is an oxygen-dependent enzyme, and its function may be limited in the anaerobic gut environment

however, oxidative stress, which is a normal process and byproduct of brain functioning, reduces this spare capacity, limiting the ability to meet heightened needs (see Supplementary Figure 2A)

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In DCM, upregulation of the copper importer SLC31A1 makes patients exceptionally sensitive to chelators, whereby a standard dose can precipitate severe deficiency [240]