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This epigenetic silencing effectively inhibits class-switch DNA recombination (CSR), somatic hypermutation (SHM), and plasma cell differentiation, thereby reducing the production of both T-dependent and T-independent autoantibodies (e.g., anti-dsDNA antibodies)

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Moreover, in pancreatic cancer, transmembrane protein 164 (TMEM164) has been shown to activate autophagy, leading to the degradation of ferritin, GPX4, and lipid droplets

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For functional medicine practitioners and researchers interested in this area, understanding what the evidence actually supports versus what remains speculative is the critical starting point