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This could lead to the prediction of future escape variants and the design of vaccine antigens that encode structural epitopes that are more difficult to escape

We don't have data on people using BPC-157 for years

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If our hypothesis is confirmed (i.e., some of these host cleavage sites prove to be functional), that leaves us with an interesting questionwhat evolutionary advantages are driving some viruses to go so far as to degrade or block the synthesis of GSH and specific host selenoproteins