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doi: 10.1038/s41416-022-01747-0 120 TianSPengPLiJDengHZhanNZengZet al

While the GHK peptide alone showed some biological activity, the copper complex demonstrated dramatically enhanced effects across multiple parameters [1]

In the absence of MTAP, the substrate methyl thionyl adenosine (MTA) accumulates and acts as a selective inhibitor of PRMT5 to inhibit PRMT5 methylation activity, 237 whereas MAT2A can produce the PRMT5 substrate SAM, 238 allowing PRMT5 to exert its oncogenic effect

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