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Heart dysfunction, lung lesions (i.e., time-dependent and time-independent features that can be acute respiratory distress syndrome exudative phase features), liver failure, and gastrointestinal lesions, widespread arterial and venous thrombosis, severe portal and caval hypertension and aortal hypotension were all counteracted [26]

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They exert their antitumor effects through multiple mechanisms, including epigenetic modification, inhibition of key signaling pathways, and modulation of the tumor microenvironment ( Figure 3 )

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