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These components are crucial in linking microbial l-tryptophan (Trp) catabolism and host endogenous Trp metabolites to regulatory T-cell function, particularly in maintaining AhR-dependent T-cell immune homeostasis at mucosal surfaces

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Literature should be read as model-specific research context, not product guidance

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ordinary least squares regression

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The results revealed that there was a similar pattern of PCNA induction by administration of BPC 157 and growth hormone, that correlated with the increase in the number of viable cells [2]

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