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LPS-induced autophagy is mediated by oxidative signaling in cardiomyocytes and is associated with cytoprotection

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However, increased NO levels could mediate the activation of Ataxia telangiectasia mutated kinase (ATM) and Liver kinase 1 (LKB1) pathways, which by stimulating AMPK resulted in mTORC1 repression and protein synthesis decline [132]