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There is evidence that the male mouse brain is more susceptible to OS due to lower levels of paraoxonase-2, which is an antioxidant enzyme positively modulated by estradiol 58

Whats more, prolonged ER stress and a defective UPR may lead to the activation of inflammatory transcriptional programs and the release of proinflammatory cytokines, generating further ER stress and oxidative stress

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It is the imbalance between the oxidants and antioxidants that result in delayed wound healing

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