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Neto FTL, Flannigan R, Goldstein M

Abstract Virus-host competition drives evolution of diverse antivirus defenses, but how they co-operate in wild bacteria and how bacteriophages circumvent host immunity remains poorly understood

Guanylate-binding proteins convert cytosolic bacteria into caspase-4 signaling platforms

This substrate site-targeting mechanism provides selectivity over related methyltransferases and other NAD salvage pathway enzymes in laboratory assay systems

These findings underscore its potential as a senolytic agent with research applications in degenerative diseases, dermatology, and regenerative medicine