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and iii) the residual phase, during which some symptoms may abate but substantial functional difficulties persist (13,14)

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15 These results support the hypothesis that NAC is capable of crossing the bloodbrain barrier and providing cysteine substrate to CNS cells, thus enhancing GSH synthesis

Administration frequency varies based on deficiency severity and individual response

[DOI] [Google Scholar] 36.Ghahremani-Majd H., Mumivand H., Khanizadeh P., Bakhshipoor F., Argento S

In PTSD mice models, trauma exposure increased tyrosine hydroxylase expression (the rate-limiting enzyme in catecholamine biosynthesis), elevating stress hormones epinephrine and norepinephrine in serum and colon, which upregulated claudin-2 expression, confirming gut barrier dysfunction (Yadav et al., 2023)