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The novel intracellular protein CREG inhibits hepatic steatosis, obesity, and insulin resistance

DOI:10.1007/ s13277-013-0778-z [ Links ] 54

A major gap in the existing literature, however, is knowledge of how specific selenoproteins can modify eicosanoid networks in such a way as to switch from a pro-inflammatory to resolution state and thereby mitigate uncontrolled inflammatory responses that lead to disease pathogenesis

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