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Furthermore, it disrupts mucosal immunity by impairing IgA plasma cell development, allowing for an increased intratumoral bacterial burden (Wang et al., 2025)

Hepatic triglycerides and lipid disposal pathways (fatty acid export and oxidation) are decreased following FABP1 ablation in fasted mice, suggesting that reduced levels of liver triglycerides are linked to a reduced hepatic lipid uptake, at least in the fasted state in which lipid flux to the liver is increased [33, 34]