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Indeed, exposure of primary mouse macrophages to such naked IONPs has identified more than 1,000 differentially expressed genes compared to untreated controls, most of them linked to inflammatory response and oxidative stress (172)

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showed that Se boosted GPX1 expression by inhibiting DNMT2-mediated DNA methylation of the GPX1 promoter from lessening ROS production in advanced glycation end-product (AGE)-induced heart failure [114]

The role of IGF1 in increasing SPC risk has been explored less

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In addition, catabolic genes Atrogin-1 and MuRF-1 were also not changed by the supplementation (data not shown)