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Sox11-modified mesenchymal stem cells accelerate cartilage defect repair in SD rats

[13] The entire clinical management of Wilsons disease is aimed at removing copper and restricting its intake, so any molecule whose function is to deliver copper systemically runs directly counter to that goal

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These disturbances, compounded by impaired mitochondrial fatty acid oxidation, ultimately contribute to dyslipidemiacharacterized by elevated serum triglycerides, LDL cholesterol (LDL-C), and total cholesterolwhich accelerates pathological processes such as atherosclerosis and fatty liver disease [119, 120]

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