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The ketogenic diet, by shifting cellular energy metabolism toward fatty acid oxidation via complex II, appears to compensate for impaired complex I activity and supports healthier mitochondrial function [96]

Each experiment was repeated five times

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The induction of GSH synthesis by nanomolar concentrations of NO in endothelial cells: a role of -glutamylcysteine synthetase and -glutamyltranspeptidase, FEBS Lett

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