Methods: Left-sided HI brain injury was induced in postnatal day 9 (P9) mice, followed by a 5 mg/kg intraperitoneal injection of 7,8-DHF
T.Picard-RieraN.Tessier-LavigneM.Baron-Van EvercoorenA.SoteloC.ChedotalA
While excess dietary Se commonly causes food poisoning complications such as diarrhea, nausea, and vomiting [77, 78]
Activated JNK translocates to mitochondria and phosphorylates anti-apoptotic Bcl-2 family members (e.g., Bcl-2 phosphorylation at serine residues such as Ser70) leading to functional inactivation of Bcl-2/Bcl-xL, and promotes pro-apoptotic conformational changes and mitochondrial translocation of Bax ( 2+ /calmodulin-dependent protein kinase II (CaMKII) via methionine oxidation (e.g., Met281/282 oxidation), producing sustained kinase activity that phosphorylates mitochondrial Ca 2+ handling proteins such as the mitochondrial calcium uniporter (MCU) or its regulatory subunits
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