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Peroxiredoxin-2: A novel regulator of iron homeostasis in ineffective erythropoiesis

The progressive decline in mitochondrial function, characterized by impaired ATP synthesis, excessive ROS production, and structural abnormalities, triggers a vicious cycle involving energy crises, oxidative stress, and inflammatory responses, ultimately accelerating cardiomyocyte death [129, 130]

therefore, they may attack RBCs, inducing membrane structure changes (erythrocyte shrinkage, membrane blebbing, and PS exposure) and, finally, eryptosis

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Here increased glutamine metabolism promoted resistance against mTOR inhibition in glioblastoma multiforme cell lines, with glutaminase expression increasing after mTOR kinase inhibition

The micro-wounds themselves cause temporary redness and sensitivity