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Previous studies have revealed that PIK3CA mutations reprogram glutamine metabolism by upregulating the expression of glutamate pyruvate transaminase (GPT) 2, thereby increasing glutamine dependency in CRC cells [101]

Therefore, both in vivo and in vitro experiments indicate that AMPK can play a role in protecting against oxidative damage, possibly by inhibiting ferroptosis to provide neuroprotection (97)

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Molecular dynamics simulations indicate that N-terminal extension stabilizes the alpha helix structure of IGF-1 LR3, enhancing its binding ability to IGF-1 receptors

Metabolic biomarkers of increased oxidative stress and impaired methylation capacity in children with autism Am J Clin Nutr December 2004 vol

[DOI] [PubMed] [Google Scholar] Khalil H, Peters M, Godfrey CM, Mcinerney P, Soares CB, Parker D (2016) An evidencebased approach to scoping reviews