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Ferroptosis has recently been identified as a regulated mode of cell death, and a growing number of studies have demonstrated that ferroptosis is closely related to the occurrence and development of various CVDs

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This occurs through the action of mobilized FAs that function as allosteric activators of uncoupling protein 1 (UCP1), the molecular mechanism for heat generation in brown fat (6, 100)

This suggests that glutathionylation of one of these cysteine residues may be an intermediate toward the formation of the intermolecular disulfide in isoforms possessing both cysteines