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doi: 10.1007/s00404-014-3279-4 89 NaykiCNaykiUGunayMKulhanMankayaMHumeyra Taskn KafaAet al

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In vitro studies suggested that bacterial components can trigger abnormal -synuclein pathology in intestinal cells directly connected with the vagus nerve [22], supporting the hypothesis of -synuclein accumulation triggered by GM changes

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