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Our data suggested that mutation of 3UTR-1 had no obvious effect to rescue the phenotype caused by DDX3X or METTL16 deficiency, whereas mutation of 3UTR-2 or 3UTR-3 fully eliminated the outcome induced by DDX3X or METTL16 inhibition (Fig
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[Drug administration through the rectum: reliability, tolerance]
Contrarily, BPC 157 counteracted prototypical muscarinic receptor antagonist atropine and mydriasis in rats and guinea pigs [86]
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