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doi: 10.1155/2022/2493053

Conversely, upregulation of the anti-apoptotic protein MCL1 has been linked to acquired resistance against sorafenib-induced ferroptosis, revealing a potential adaptive mechanism (130)

However, ferrostatin-1 and rhosin effectively reversed DMM-associated changes in MDA levels, SOD and GSH activity and T-AOC (Fig

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