The ins and outs of mitochondrial iron-loading: the metabolic defect in Friedreichs ataxia [J]
Before the target cells can use the fatty acids for ATP production and oxidation, the fatty acids with chain lengths of 14 or more carbons must be activated and subsequently transported into mitochondrial matrix of the cells in three enzymatic reactions of the carnitine shuttle
In this regard, exercise could enforce improvements of functions in other organs by ameliorating muscle metabolism and counteracting atrophy
unraveled the function of GSH in keratinocytes and showed that GSH deficiency affects keratinocyte survival and wound repair
10.1093/burnst/tkac059 120
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