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doi: 10.1213/ane.0000000000004529 7 Bello-MedinaP

About this article Cite this article Zhang, H., Ding, X., Hu, X

Excessive glutamate release activates NMDARs and AMPARs, leading to Ca 2+ overload, promoting neuronal oxidative stress, inducing mitochondrial damage, and increasing the risk of cell death

Nitro group is also known to quench fluorescence of the attached fluorophore because of its strong electron withdrawing nature, however fluorescence can be restored upon its reduction to amine

Regulatory status (US) Not FDA-approved research-use Compound Benzodiazepines What it is Prescription drugs Mechanism (in plain English) GABA-A agonists

The evidence is diverse and includes extensive work with laboratory animals, otherwise unexplained associations, factors associated with APAP metabolism, and limited studies in humans