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doi: 10.1172/jci.insight.173205 12 DiricanO.KayginP.OguztznS.HusseiniA

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Mechanistically, the upregulation of OCTN2 promotes the proliferation and migration of HCC cells in vitro and augments the growth and metastasis of HCC, as well as the cancer stem-like properties of HCC by increasing FAO and OXPHOS, which depends on PGC-1 signaling

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Binding to the IGF1R initiates intracellular signaling

Oxidative stress stimulates invasive potential in rat C6 and human U-87 MG glioblastoma cells via activation and cross-talk between PKM2, ENPP2 and APE1 enzymes