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Subsequent sections examine the core mechanismsanti-apoptotic, angiogenic, antioxidative, immunoregulatory and epigeneticthrough which MSCs reprogramme the damaged ovary, and we evaluate complementary strategies such as cell-free exosomes, small-molecule pre-conditioning and bio-engineered artificial ovaries

This hypothesis, while speculative, could explain the peptides ability to normalize both deficiency and excess states across diverse neurotransmitter disturbances

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