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VMAT2 mediates the sequestration of monoamine neurotransmitters, including dopamine and norepinephrine (NE), into synaptic vesicles

Collectively, these results suggested that EV-derived VDAC1 protein is a potential therapeutic target for averting PTX resistance of GC and a specific inhibitor of VDAC1, such as DIDS, might serve as a component of combination therapy to overcome PTX resistance

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Goel can deliver protocols at this depth without diluting the work

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TB-500 moves cells into those vessels), they are rarely researched in isolation anymore