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Ullas Kamath et al., 2019

This bidirectional regulation establishes a self-perpetuating cycle where initial ferroptotic events trigger inflammatory responses, which in turn promote additional rounds of ferroptosis in surrounding tissue

M.TsaiK

Pericyte deficiency intensifies amyloid-beta (A) and tau pathology and accelerates neuronal loss in the absence of A-precursor protein, leading to cognitive decline [45]

Additionally, BPC-157 interacts with the dopaminergic and serotonergic systems, which may explain its observed neuroprotective properties in preclinical models of traumatic brain injury and nerve damage

10.1038/s41587-019-0233-9 159 WilsonI