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By binding to G-actin, TB-500 maintains a pool of actin monomers that prevent the premature hardening of the cellular skeleton

doi: 10.7860/JCDR/2013/6635.3732 122

Evidence from animal models points toward support of axonal continuity, synaptic stability, and functional recovery following nervous system injury

Supports the reduction of systemic and localized inflammation without suppressing immune function

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Moreover, these data reinforce that the expedited loss of Fe-S protein function in the absence of CHAC1 is associated with a deficiency in de novo cluster synthesis