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The fact that 75% of smokers do not develop COPD points to strong defense mechanisms to handle the increased oxidant burden that smoking puts on the lung

pneumoniae , and to compare these findings with those reported for ahpD [29] and tpxD [7], we constructed deletion mutants for each gene in the R801 strain, and we determined H 2 O 2 susceptibility expressed in percentage of bacterial survival that survived at 20 mM H 2 O 2 for 30 min [7]

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See also References Van Konynenburg, Rich (September 19, 2009)

MMR, mismatch repair