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50 More and more evidence shows that the abnormalities in intercellular communication within the pancreatic microenvironment play a critical role in the pathophysiological response of the pancreas to various stimuli
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13a, Supplementary Table 9), suggesting that the distinct rotamer orientation of H296 ECL2 in Cagri-bound CTR (to accommodate the Glu linker) and/or the observed more open ECL3 conformation induced by the lipidated peptide may facilitate or stabilize an active CTR conformation (Supplementary Fig
doi: 10.3389/fimmu.2020.01027 221 YinYChoiS-CXuZPerryDJSeayHCrokerBPet al
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