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In other words, in addition to inhibiting the expression of p-STAT3, GHK-Cu likely targets other pathways that inhibit the expression of inflammatory factors

By stimulating angiogenesis and encouraging the infiltration of macrophages and fibroblasts, BPC-157 emerges as a promising therapeutic tool for managing various types of wounds

Importantly, its mechanism is complementary to GLP-1 receptor agonists, making it an attractive candidate for combination therapy

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