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In patients with enthesitis or axial disease refractory to NSAID therapy, there is little evidence for benefit with nonbiologic DMARDs, so that TNF inhibitor therapy may be warranted in this situation

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Mechanistically, HGF-induced synapse formation is mediated by PI-3K and not MAPK activation, as inhibition of the PI-3K/AKT pathway affects synaptogenesis, while blocking MAPK/ERK does not decrease synapse density and only affects dendritic growth (Eagleson et al., 2016)

The goal is to maintain stable levels of the peptide in the system during the research period

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