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Multiple components of cigarette smoke conspire to harm endothelial cells, and damaged endothelial cells lay the groundwork for formation of cholesterol-laden, inflammatory plaque

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Bonnie Herzog, a tobacco stock analyst at Wells Fargo Securities, estimated last year that 2013 e-cigarette sales would exceed $1 billion in the U.S

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Although 12 of 15 studies in which the effects of smoking cessation on vascular endothelial function was assessed by physiological methods including measurements of FMD and reactive hyperemia index (RHI) showed that smoking cessation improved FMD, RHI or vascular response to vasoactive agents [18, 21, 22, 25,26,27,28,29, 32,33,34,35], three of those 15 studies showed no effects of smoking cessation on FMD or RHI [89,90,91]