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Egan SK, Bolger PM, Carrington CD

This trial used two animal models of MS: the experimental autoimmune encephalomyelitis, EAE, model to mimic the autoimmune response and neurodegeneration in MS and the cuprizone, CPZ, model to mimic demyelination to nerve cells and to study remyelination of the damaged nerve cells in MS

Biochim Biophys Acta Mol Basis Dis 1863(5):10661077

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View at Publisher | View at Google Scholar Enebo LB, Berthelsen KK, Kankam M, Lund MT, Rubino DM, et al

This instability of heterochromatin structure is closely associated with cancer development and could be partly repaired by histone methyltransferase SETDB1 that is specifically recruited to the UV-damaged heterochromatic regions and maintains the H3K9me3 modification on newly deposited H3 histones, helping to protect the cell from the effects of genomic instability [153]