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NRF2-dependent survival in T-B2B cells is mediated by uncoupling stress from the AKTGSK3 axis Our previous data indicated that chronically smoke-exposed cells acquire stress-resistant phenotypes characterized by the constitutive activation of NRF2

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In line with these findings, use of a 3D epigingival tissue model exposed to e-cigarette found IL-8 and RAGE to be elevated, but only PGE2 was found to be elevated significantly (91)