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This is clearly demonstrated by experiments in which the administration of the neurotoxin MPTP, which blocks Complex I of the mitochondrial electron transport chain, induces characteristic features of PD in mice [56, 57]

now with Dalhousie University Department of Medicine, Halifax, Nova Scotia, Canada Michael D

Sugii S, Ohishi I, Sakaguchi G

Sufficient NAD+ availability is necessary for these enzymes to operate, so raising NAD+ levels could in theory modulate pathways related to aging, inflammation, and cell survival

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